,1,The 2022 Monkeypox outbreak has different signs and symptoms compared to previous years, and compared to infections that are in endemic regions.
Monkeypox usually causes a specific rash after the prodromal symptoms, like severe fatigue, fevers, chills, and body aches, with swollen lymph nodes. But with this monkeypox outbreak, some patients have had genital, rectal, and/or oral lesions, but without the initial prodromal symptoms.
If there is a prodromal period, it typically lasts up to five days.The rash usually starts within 1 to 4 days of after the fever, and lasts for about two to three weeks, although the rash does sometimes appear without the prodromal symptoms.
The number of skin lesions can be anywhere from just a few, to a few thousand.
And in severe cases, they can coalesce until large sections of skin slough off.
The rash tends to involve the face, and can also involve the palms of the hands and soles of the feet.
It can also be in the mouth and throat, the inside of your eyelids, and genital areas.
In fact, with this outbreak, some patients had proctitis, meaning inflammation of the prostate gland. In some cases the only lesions found were those in the genital regions.
Lesions progress through several stages:
The painful skin lesions typically start out as 2 to 5 mm diameter spots, and then evolve into papules, then vesicles, and later pustules. They eventually crust over and fall off, about 1-2 weeks after the rash first starts.
Complications
Sometimes monkeypox can cause complications, like secondary bacterial infections, pneumonia, sepsis, and infection of the cornea of the eye, subsequent loss of vision.
During the current outbreak there have been a few unusual presentations.
For instance, some people started with the rash in the genital areas, with later spread to the face and trunk.
In other instances, the lesions never spread to the face or arms or legs.
Patients have also presented with anal and rectal pain, tenesmus, bleeding from the rectum, and tenesmus, which is the feeling that you need to pass stools, even though your bowels are already empty.
Can you die from Monkeypox?
For most people, monkeypox is a self-limited disease with the symptoms lasting 2-4 weeks. The severity of disease depends on age, and underlying health status. Children are more likely to have severe disease compared with adults. The mortality associated with monkeypox has varied. In Central Africa, where they have the worse strain, the fatality rate was around 10%. So far, with this outbreak, there haven’t been any reported deaths in nonendemic countries.
How to diagnose Monkeypox
If the diagnosis of monkeypox is suspected, a lesions will need to be swabbed for PCR testing.
Blood sample testing for monkeypox virus can be used to help make the diagnosis of well, especially if viral testing is not able to be performed.
Patients with monkeypox typically have detectable levels of anti-orthopoxvirus IgM antibody during the period of 4 to 56 days after rash onset.
Treatment
Most infections are mild don’t require treatment in a hospital. Other than medical care, these people should stay at home.
Unexposed people shouldn’t be in the home of an infected person, if possible.
If you’re infected, and you have to be around others, the skin lesions should be covered with clothing to minimize risk of contact with monkeypox lesions, as well as wearing a mask.
Household members providing care to patients with monkeypox should use disposable gloves for direct contact with lesions, and washing of hands afterwards.
You’ll also have to be mindful that clothes, bedsheets, and towels can become contaminated, and will need to be washed accordingly.
#monkypox #covid #virus
,1,How do you get rid of the flu in 24 hours? For most healthy people, the flu is an uncomfortable but short-term illness that resolves itself as the immune system fights it off. Symptoms usually appear from one to four days after exposure to the virus, and they last five to seven days
#flu #health #recovery
,1,Monkeypox is spreading around the globe in a way that hasn't been spread before. Scientists at CDC collaborate with the Massachusetts Department of Public Health officials to investigate a situation in which a U.S. resident tested positive for monkeypox on May 18 after returning to the U.S. from Canada. They’re also tracking multiple clusters of monkeypox cases reported this month in several countries that don’t usually report monkeypox, including Europe and North America. This is why it becomes such a concern.
It’s not clear how people in those clusters were exposed to the monkeypox virus, but some of the known causes include people who self-identify as men who have sex with men.
So let's jump into what monkeypox is and the most important things you need to know – what causes it, how to prevent it, the symptoms, and its treatment. Monkeypox is an orthopoxvirus, and it was first isolated in the late 1950s from a colony of sick monkeys. The virus itself is basically the brother of the smallpox virus, but person-to-person spread and mortality are MUCH smaller compared to smallpox. It is widely believed that the monkeypox virus has infected humans for thousands of years in sub-Saharan Africa but wasn’t actually identified as a cause of disease in HUMANS until the 1970s in the Democratic Republic of the Congo. Of all the reported 59 cases in the 1970s, 17% of them died.
How did these individuals get it?
They were exposed to small forest animals, like rodents, squirrels, and monkeys.
With the African outbreak from 1996 to 1998, the mortality rate was 5%. The first outbreak of monkeypox in the Western Hemisphere occurred in the United States in 2003. This virus is typically acquired through contact with an infected animal's bodily fluids or through a bite. Monkeys and humans are incidental hosts. The most likely culprit animal for being the reservoir host…rodents. Including prairie dogs.
That US outbreak in 2003 consisted of 71 cases in the US; they identified and traced the infection back to prairie dogs, which appeared to have acquired the virus from African rodents when the two species were housed at a distribution center in Illinois. It’s also important to note that a complex exposure, such as a bite wound from an infected animal, is more likely to cause severe infection than a non-complex exposure, such as simply touching the prairie dog. Although person-to-person transmission could not be excluded, most human cases had direct exposure to animals.
But unfortunately, Human-to-human transmission can also occur. One way is via large respiratory droplets. But unlike COVID, the virus isn’t really good at spreading this way. For example, droplet transmission likely requires prolonged face-to-face contact for 3+ hours. This virus is more likely to spread when close contact with infectious skin lesions, especially during sex.
All in all, most of those cases were identified in men who have sex with men. So what happens if you get the virus? The incubation time, meaning the time of exposure to the time of developing the illness, is about 12 days….but most monkeypox infections are asymptomatic. If you do get symptoms, you’re looking for fevers, chills, muscle aches, and swollen lymph nodes, with the famous painful, non-itchy rash you see in all the pictures. Typically, about 2 days after the fever starts, the rash appears on the chest and back and spreads outward to the palms and soles of the feet. The lesions are around 0.5 to 1 centimeter in size. They start out as spots and then progress over the next few weeks to form vesicles and pustules, and in the end, they scab up and slough off.
Doctor Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
Website: https://doctormikehansen.com/
Contact & Social Media Links: https://doctormikehansen.com/contact/
#monkeypox
0:00 Introduction
0:30 MONKEYPOX IS ONLY ENDEMIC TO PARTS OF AFRICA
1:12 MONKEYPOX IS MUCH MORE BENIGN COMPARED TO SMALLPOX
1:41 59 CASES THERE IN THE '70S
1:54 AFRICA OUTBREAK 1996-1998
3:44 SMALLOX WAS ERADICATED IN 1980
4:06 2005-2007- 760 CASES
4:23 5X LOWER RISK IF THEY RECEIVED SMALLPOX VACCINE
6:09 INCUBATION -12 DAYS
8:11 TECOVIRIMAT
8:16 TRANSLATION: IT PREVENTS THE VIRUS SPREAD IN THE BODY
8:27 SMALLPOX VACCINE PROTECTS AGAINST MONKEYPOX
8:35 MODIFIED VACCINIA ANKARA (MVA)
,1,Doctor Rips Joe Rogan after taking Ivermectin for COVID
Joe Rogan Got COVID. He took "the kitchen sink" for it, including ivermectin. He was also thankful for "Modern Medicine." This is, of course, after he claimed that all you need is "natural immunity."
In this video, I'm calling out Joe Rogan. This is what "Modern Medicine" has to say in response.
Also, I refer to a bunch of videos within this video, including ivermectin. Be sure to check out my other videos for more depth on COVID.
This channel is about accurate health information and was created to battle misinformation even before COVID came along.
Doctor Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
Website: https://doctormikehansen.com/
Doctor Hansen's Courses: https://doctormikehansen.com/courses/
Contact and Social Media Links: https://doctormikehansen.com/contact/
#ivermectin #covid #joerogan
,1,Woman Dies 4 days after getting COVID Vaccine | Post Vaccine Deaths
Kassidi Kurill, a 39-year-old Single Mom. Her family describes her as healthy, happy, and "having more energy" than most. She had no known health problems. Unless you count Trigeminal Neuralgia. She took Botox for that. She also took Birth Control. She worked as a surgical tech for local plastic surgeons. She gets the Moderna Covid vaccine. Her first covid shot goes without incident. Other than a sore arm. Second dose of covid vaccine Moderna - not so much. She gets her 2nd Covid shot on Feb. 1 Monday. Later that evening, she had a sore arm, nausea, and fever. That's not unheard of after someone gets the covid vaccine. Some people get a sore arm, fatigue, body aches, slight fever, and chills. Especially after the second covid shot. Especially for younger people. When this happens, people can take Tylenol or ibuprofen.
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This is her GoFundMe page:
https://www.gofundme.com/f/kassidi-kurill-medical-and-funeral-expenses
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No big deal, right? Unless they take too much.
She did take Tylenol, aka Acetaminophen, but we don't know how much. She felt a little better the next day. By Wednesday, she could not pee despite drinking lots of fluids. She says it's the worst she ever felt. Thursday, 6 am, she tells her parents she needs to go to the hospital. In the ER, she immediately starts throwing up. Doctors say, "she was very, very sick, and her liver was not functioning." Blood tests show that her liver is not working. The doctors give her IV fluids. They also give her a drug to counteract the effects of Tylenol on the liver. It's called NAC, or n-acetylcysteine. But the damage was already done. Around this time, she becomes incoherent. Acute liver failure causes the brain to swell, called cerebral edema. If severe enough, it can result in a coma. At this point, her only chance of survival is with a liver transplant.
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Doctor Mike Hansen's Programs ⏩
FULL Course for MORE ENERGY:
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So she was flown to a transplant hospital. But when the liver completely fails, it causes other organs to follow suit. Especially the kidneys and brain, and soon after heart and lungs. She's put on full life support, including a breathing tube. Even if there was a match for a new liver, she is too unstable to undergo that operation. She dies. About 30 hours after she first arrived at the ER. She leaves behind a young daughter who wants people to hear her story.
A lot of people will conclude that the covid vaccine caused her death.
But timing by itself does not prove causation.
And many people want to know, did the covid vaccine cause her death?
And if not, then what?
A Utah medical examiner completed a full autopsy...but the results remain private. The Medical Examiner said the autopsy did not show a link between the covid vaccine and death.
Which is hardly surprising. If a covid vaccine caused someone to die shortly after receiving it, how would it do so? The only medical explanation would be an immunological reaction. Meaning an allergic reaction. So far in the US, there have been over 200 million covid vaccine doses given. No one has died from a severe allergic reaction as a result of the COVID vaccine.
And in her case, it's obvious that she did not have an allergic reaction. For example, there was no swelling in the face or tongue or tightening of the throat. Deaths after COVID vaccinations are reported to the Vaccine Adverse Event Reporting System.
Between Dec. 14 and mid-March, over 109 million covid vaccine doses in the US.
Of those, there have been less than 2,000 reports of death on the VAERS system, which is 0.0018%. A reported death in the VAERS system doesn't mean the covid vaccine caused it.
So the big question in Kassidi Kurill's case is…what caused her acute liver failure?
In the US, Acetaminophen accounts for 40% of cases.
In 18% of cases, there is no identifiable cause. However, 13% are due to idiosyncratic drug reactions.
The general recommended dose for Acetaminophen in healthy people is no more than 4 grams per day. However, sometimes people end up taking more than they should. This accounts for almost half of the cases of acetaminophen-induced acute liver failure.
Doctor Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
Website: https://doctormikehansen.com/
Doctor Hansen's Courses: https://doctormikehansen.com/courses/
Contact and Social Media Links: https://doctormikehansen.com/contact/
#covidvaccine #vaccine #covid
,1,12 COVID Autopsy Cases Reveal the TRUTH "HOW COVID PATIENTS DYING"
In all 12 COVID Autopsy cases, the cause of death was found within the lungs or the pulmonary vascular system. Those who did not die of large pulmonary emboli died of extensive inflammation, meaning pneumonia with ARDS. In these COVID Autopsy cases, the lungs were wet and heavy, like a saturated water sponge. The lung surfaces often had a distinct patchy pattern, with pale areas alternating with slightly protruding and firm, deep reddish-blue Hypercapillarized areas.
This indicates areas of intense inflammation, with endothelial dysfunction that can be seen at the microscopic level. When they looked at the lungs' slices under the microscope, they found diffuse alveolar damage in 8 COVID Autopsy cases. Specifically, they saw hyaline membrane formation, tiny clots in the capillaries, capillaries engorged with red blood cells, and other inflammatory findings. All these findings represent ARDS. They also found lymphocytes, a type of white blood cell, infiltrated these areas of infiltration. This fits the picture of viral pathogenesis.
⏩ Timestamps
00:00 - Start
02:38 - The starting point of the COVID Autopsy Analysis
04:10 - Why we get COVID false Negative Test
04:34 - Rest of the Part of COVID Autopsy Analysis
09:45 - Big Takeaway's from the Findings in this COVID Autopsy Study
12:30 - Minimize the chances of having the severe disease if you were to get COVID
This is the link to the main study in this video:
https://www.acpjournals.org/doi/10.7326/M20-2003
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Doctor Mike Hansen's Programs ⏩
FULL Course for MORE ENERGY:
https://learn.doctormikehansen.com/products/increase-energy-course/
FREE Course for MORE ENERGY:
https://doctormikehansen.com/6-days-to-more-energy/
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They also looked at the pharynx of these COVID patients, meaning in their throat. The lining of the throat, or mucosa, was hyperemic, meaning very red and irritated. They saw lymphocytes invading at the microscopic level, which is consistent with a viral infection. In one COVID case, a COVID patient had lymphocytes invade his heart muscle, findings that are consistent with what we call viral myocarditis. More than half of the COVID patients had large blood clots in this study. One-third of the COVID patients had pulmonary embolism as the direct cause of death.
All the others died of intense inflammation in their lungs related to pneumonia with ARDS (Acute Respiratory Distress Syndrome). Recently, studies show that about 1/3rd of COVID patients with severe COVID have blood clots. In another study of 191 COVID patients, half of those who died had clots, compared with 7% of survivors. And levels of D-dimer that were greater than 1000 µg/L were associated with a fatal outcome. So it's pretty clear now that the COVID has caused many clots to form in moderate to severe COVID disease.
How is this happening?
It's likely a combination of reasons that have to do with downregulation of the ACE2 receptor in the lung alveoli, with a subsequent shift towards having more angiotensin II in the lungs, and less angiotensin 1-7 and 1-9 in the lungs, and when this happens, this leads to more cytokine storm with more inflammation, more constriction of pulmonary arteries, and more clots that develop. That, in turn, leads to more endothelial dysfunction in the capillaries surrounding the alveoli. Also, there is evidence that the virus attaches to the ACE2 receptors of those endothelial cells that line those capillaries, which further propagates inflammation and clotting.
And in the cytokine storm that develops there, RANTES, a chemokine, binds to the CCR5 receptor of CD4 and CD8 lymphocytes, and that causes those lymphocytes to infiltrate those areas of inflammation and, in doing so, further contributes towards the inflammatory reaction. This is why we see low levels of CD4 and CD8 lymphocytes in severe COVID. Endothelial damage can also lead to the development of antiphospholipid antibodies, which are harmful because they trigger blood clot formation. That's why COVID patients with clots with antiphospholipid antibody syndrome need to be on blood thinners.
Doctor Mike Hansen, MD
Internal Medicine | Pulmonary Disease | Critical Care Medicine
Website: https://doctormikehansen.com/
Doctor Hansen's Courses: https://doctormikehansen.com/courses/
#covid #autopsy
